0601. An inhibitor of pro-inflammatory enzyme soluble epoxide hydrolase improves hippocampus dependent memory function after cardiac arrest and cardio-pulmonary resuscitation in mice

نویسندگان

  • T Fujiyoshi
  • S Hoka
چکیده

Introduction Promotion and improvement of cardiopulmonary resuscitation (CPR) technique contributes to increase of the survivors from unexpected cardiac arrest (CA). However, many patients have some neurological deficits, because of few effective treatment to improve the outcome. Hippocampus damage causes a memory dysfunction which suffers the survivors. Microglia activation caused neuron damage following brain stroke and inhibition of proinflammatory soluble epoxide hydrolase (sEH) reduced the infarction size in partial ischemia mice model.

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Inhibition of soluble epoxide hydrolase after cardiac arrest/cardiopulmonary resuscitation induces a neuroprotective phenotype in activated microglia and improves neuronal survival.

Cardiac arrest (CA) causes hippocampal neuronal death that frequently leads to severe loss of memory function in survivors. No specific treatment is available to reduce neuronal death and improve functional outcome. The brain's inflammatory response to ischemia can exacerbate injury and provides a potential treatment target. We hypothesized that microglia are activated by CA and contribute to n...

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عنوان ژورنال:

دوره 2  شماره 

صفحات  -

تاریخ انتشار 2014